Electrocardiogram (ECG) interpretation is a critical skill in Advanced Cardiovascular Life Support (ACLS). The ability to recognize life-threatening rhythms helps guide timely and appropriate interventions during cardiac emergencies.
Why It’s Important:
ECG interpretation directs ACLS interventions (shock vs. no shock, pacing vs. medications).
Helps differentiate between stable and unstable rhythms.
Identifies reversible causes of cardiac arrest (Hs & Ts).
P Wave: Atrial depolarization (contraction of the atria).
QRS Complex: Ventricular depolarization (contraction of the ventricles).
T Wave: Ventricular repolarization (recovery phase).
PR Interval: Time for electrical impulse to travel from the atria to the ventricles.
QT Interval: Total time for ventricular depolarization and repolarization.
Key Takeaway: Understanding these ECG components helps distinguish normal vs. abnormal rhythms!
HR: 60-100 bpm
Rhythm: Regular
P Wave: Present before each QRS
PR Interval: Normal (0.12-0.20 sec)
QRS Complex: Narrow (<0.12 sec)
Management: No intervention required.
Rhythm: Regular
P Wave: Normal
PR Interval: Normal
QRS Complex: Normal
Management (If Symptomatic)
Key Takeaway: Treat only if symptomatic (hypotension, altered LOC, shock).
P Wave: Normal
PR Interval: Normal
QRS Complex: Normal
Management:
Key Takeaway: Sinus tachycardia is usually compensatory—fix the cause, don’t just lower the HR!
Rhythm: Irregularly irregular
P Waves: Absent (fibrillatory waves)
QRS Complex: Normal (but irregular R-R intervals)
Management:
Key Takeaway: AF increases stroke risk—assess for anticoagulation!
Rhythm: Regular or irregular
P Waves: Sawtooth flutter waves (F-waves)
QRS Complex: Normal
Management:
Key Takeaway: Atrial flutter can rapidly degenerate into AF—manage similarly!
HR: >100 bpm
QRS Complex: Wide (>0.12 sec)
Types: Monomorphic (uniform QRS) or Polymorphic (varied QRS, e.g., Torsades de Pointes).
Management:
Pulseless VT → Treat as Cardiac Arrest (CPR + Defibrillation).
Stable VT (with pulse) → Antiarrhythmic therapy (Amiodarone 150 mg IV over 10 min).
Unstable VT (Hypotension, shock) → Immediate Synchronized Cardioversion (100 J).
Key Takeaway: Always check for a pulse—Pulseless VT requires immediate defibrillation!
Chaotic, disorganized ventricular rhythm.
No identifiable P waves, QRS complexes, or T waves.
Patient is pulseless and unresponsive.
Management:
Immediate High-Quality CPR + Defibrillation (120-200 J biphasic).
Epinephrine 1 mg IV every 3-5 min.
Amiodarone 300 mg IV (if refractory VF after 3 shocks).
Key Takeaway: VF is ALWAYS a cardiac arrest rhythm—defibrillate ASAP!
No P waves, QRS complexes, or organized activity.
Patient is pulseless and unresponsive.
Management (Non-Shockable Rhythm)
Key Takeaway: Asystole = Poor survival rates. Focus on CPR & identifying reversible causes!
Organized ECG activity (normal or abnormal rhythm) but NO pulse.
Management (Non-Shockable Rhythm)
Key Takeaway: Electrical activity ≠ perfusion. Always check for a pulse!
Algorithm | ECG Role |
---|---|
Cardiac Arrest | Determines shockable (VF/pVT) vs. non-shockable (PEA/Asystole) rhythms. |
Bradycardia | Identifies HR <60 bpm & guides pacing/medication use. |
Tachycardia | Differentiates between narrow vs. wide QRS & stable vs. unstable tachycardia. |
Key Takeaway: ECG interpretation directs ACLS interventions—treatment is rhythm-specific!
Recognize shockable (VF/pVT) vs. non-shockable (PEA/Asystole) rhythms.
Identify life-threatening bradycardia & tachycardia.
Use ECG to determine need for cardioversion, defibrillation, or pacing.
Always correlate ECG findings with the patient’s clinical status!
Takeaway: ACLS ECG interpretation is a critical skill—quick recognition leads to life-saving interventions!